Abdulaziz Issa

Generally, cancer is genetic malfunction occurs to a cell which leaves the cell uncontrolled in terms of division. In other words, it leaves it in a state of metosis splitting very fast and not exactly normal, as if the cell has gone crazy and splitting at a terrible speed, so it leads to a tumor in the place of the infected cell, and it is not enough. This is a procedure called Metastasis, which is the transfer of cancerous cells from an organ. For another member, and this operation is done via The Blood or the Lymphatic System Cancer is the second-leading cause of death globally and is responsible for about 9.6 million deaths in 2018 and smoking is responsible for 22% of cancer deaths and 10% due to obesity or malnutrition and drinking alcohol. Based on the Warburg Effect study, which her idea is about consuming glucose cancer cells in larger quantities than nature to grow and this study took 3 years, and cells as they know to divide need energy, which is taken by glucose, amino acids and protein. And the fats. Warburg scientist observed a phenomenon in tumors that cancer cells tend to analyze sugar primarily for energy and assume that the dysfunctional mitochondria is the major cause of the higher percentage of sugar breakdown and may be the cause of tumor development. Currently, in this research, they are working on the production of a drug that makes infected cells unable to take their full needs of glucose in the blood But up to now there is no study that has proven that in a certain protocol for diabetes patients follow, the patient can stabilize or treat the tumor But in general, if the glucose level is high in the blood, it leads to accelerating the division of infected cells, and on the same note, low glucose level in the blood has been associated with cancer patients with a more positive outcome

In general, adducin is membrane skeleton protein which consists of either A- and B- or A- and Y-subunits, which to a large extent are similar in amino acid sequence and domain organization. The selective beneficial effect of diuretics over the other drugs was not present in carriers of the Gly/Gly ADD1 genotype. Manunta, et al. 2007 reported in his review articles on adducing polymorphisms and hypertension that selective beneficial effect of diuretics over the other drugs in carriers of the 460Trp ADD1 allele support the notion that matching of the genetic mechanism with the drug mechanisms of action produces a clear benefit probably because the magnitude of the counter regulatory mechanism and, hence, the global cardiovascular risk may be minimized.